Proteasomal degradation of Saccharomyces cerevisiae Sup35p fibrils (negative‐stained electron micrograph in the background) generates amyloidogenic peptides derived from its N‐terminal prion domain (leading to the [PSI+] phenotype). For further details readers are referred to the article by Kabani et al. on pp. 507‐528 of this issue.

Publisher： John Wiley & Sons Inc

E-ISSN： 1365-2958|92|3|i-i

ISSN： 0950-382x

Source： MOLECULAR MICROBIOLOGY, Vol.92, Iss.3, 2014-05, pp. : i-i

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A role for the proteasome in the turnover of Sup35p and in [PSI+] prion propagation

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