Length-dependent gametic CAG repeat instability in the Huntington's disease knock-in mouse

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Human Molecular Genetics, Vol. 8, Iss. 1, 1999-01 ,pp. : 115-122

Oxford University Press

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Nuclear-targeting of mutant huntingtin fragments produces Huntington's disease-like phenotypes in transgenic mice

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HD CAG repeat implicates a dominant property of huntingtin in mitochondrial energy metabolism

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Accumulation of N-terminal mutant huntingtin in mouse and monkey models implicated as a pathogenic mechanism in Huntington's disease

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Absence of Disease Phenotype and Intergenerational Stability of the Cag Repeat in Transgenic Mice Expressing the Human Huntington Disease Transcript

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