APC mutations in colorectal tumours from FAP patients are selected for CtBP-mediated oligomerization of truncated APC

By Schneikert Jean   Brauburger Katharina   Behrens Jürgen  

Human Molecular Genetics, Vol. 20, Iss. 18, 2011-09 ,pp. : 3554-3564

Oxford University Press

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APC mutations in FAP-associated desmoid tumours are non-random but not ‘just right’

By Latchford Andrew   Volikos Emmanouil   Johnson Victoria   Rogers Pauline   Suraweera Nirosha   Tomlinson Ian   Phillips Robin   Silver Andrew  

Human Molecular Genetics, Vol. 16, Iss. 1, 2007-01 ,pp. : 78-82

Oxford University Press

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The ‘just-right’ signaling model: APC somatic mutations are selected based on a specific level of activation of the  -catenin signaling cascade

By Albuquerque Cristina  

Human Molecular Genetics, Vol. 11, Iss. 13, 2002-06 ,pp. : 1549-1560

Oxford University Press

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Familial Infiltrative Fibromatosis (Desmoid Tumours) (MIM135290) Caused by a Recurrent 3′ APC Gene Mutation

By Scott Rodney J.   Froggatt Nicola J.   Trembath Richard C.   Evans D. Gareth R.   Hodgson Shirley V.   Maher Eamonn R.  

Human Molecular Genetics, Vol. 5, Iss. 12, 1996-12 ,pp. : 1921-1924

Oxford University Press

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Truncated APC regulates the transcriptional activity of -catenin in a cell cycle dependent manner

By Schneikert Jean   Grohmann Annette   Behrens Jrgen  

Human Molecular Genetics, Vol. 16, Iss. 2, 2007-01 ,pp. : 199-209

Oxford University Press

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